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Antifungal drugs (with mechanism of action) mnemonic

usmle1mikmonics:

Love it IkaN! Here’s a few more (including ones from the pharMnemonics cards series and a bunch were from one source online but I can’t remember which, sorry!)

Azoles:

Fluconazole - Fluid Fluco flows into CNS (used for cryptococcal meningitis)

Clotrimazole - Cutaneous (topical) (also Mi

(Source: medicowesome.blogspot.in)

(Source: ecgmadesimple.com)

What is Acute or Subacute Bacterial Endocarditis?

mynotes4usmle:

sidratime:

Acute or Subacute Bacterial Endocarditis is an infection of the heart’s endocardium. The endocardium is the inner lining of the heart muscle, which also covers the heart valves. Bacterial Endocarditis can damage or even destroy your heart valves. The difference between acute and subacute bacterial endocarditis is acute bacterial endocarditis is a sudden onset, whereas subacute bacterial endocarditis is a gradual onset.

Acute endocarditis most often occurs when an aggressive species of skin bacteria, especially a staphylococcus (staph), enters the bloodstream and attacks a normal, undamaged heart valve. Once staph bacteria begin to multiply inside the heart, they may send small clumps of bacteria called septic emboli into the bloodstream to spread the infection to other organs, especially to the kidneys, lungs and brain. Intravenous (IV) drug users are at very high risk of acute endocarditis, because numerous needle punctures give aggressive staph bacteria many opportunities to enter the blood.If untreated, this form of endocarditis can be fatal in less than six weeks.

Subacute endocarditis is caused by one of the viridans group of streptococci (Streptococcus sanguis, mutans, mitis or milleri) that normally live in the mouth and throat. Streptococcus bovis or Streptococcus equinus also can cause subacute endocarditis, typically in patients who have some form of gastrointestinal cancer, usually colon cancer. Subacute endocarditis tends to involve heart valves that already are damaged in some way, and it usually is less likely to cause septic emboli than acute endocarditis. If untreated, subacute bacterial endocarditis can worsen for as long as one year before it is fatal.

Acute: no previous heart valve damage

  • S. aureus (for non IVDA and IVDA)

Subacute: previous heart valve damage

  • Viridans group: S.mutans, S.sanguis; with hx of dental work without prophylaxis.
  • Group D strep: S. bovis, Enterococcus; with hx of GI or GU qx without prophylaxis
  • S. epidermidis, Candida, Aspergillus, Pseudomona, Viridans group: IVDA

Dx: Dukes criteria

(Source: jmarkmoralesmd.com)

usmlepathslides:

Abruptio placentaThis slide shows multiple sections through placenta with a retroplacental clot.  Abruptio placenta will present with painful vaginal bleeding and it represents a premature separation of the placenta producing a retroplacental clot.  It’s a complication of pre-eclampsia and any cause of hypertension.  It is associated with cocaine addiction.

usmlepathslides:

Abruptio placenta
This slide shows multiple sections through placenta with a retroplacental clot.  Abruptio placenta will present with painful vaginal bleeding and it represents a premature separation of the placenta producing a retroplacental clot.  It’s a complication of pre-eclampsia and any cause of hypertension.  It is associated with cocaine addiction.

Jul 5
Tidal percussion

Tidal percussion

Microbiology Genetics

itisoktobesmart:

  1. Transformation
  2. Conjugation:  F+ —> F-  &  Hfr —> F-
  3. Transposition 
  4. Transduction: Generalized   &  Specialized

(Source: sidratimes)

Slipped disc

Slipped disc

BASIC MOTOR PATHWAY

Corticospinal tracts

sub arachnoid hemorrhage

sub arachnoid hemorrhage

medicowesome:

Minimum alveolar concentration, blood gas partition coefficient, AV gradient of anaesthetics simplified

Hi!
This is what I understood. I hope it helps you understand as well =)

I talk about minimum alveolar concentration, blood gas partition coefficient, AV gradient of anaesthetics, rise in partial pressure, lipid and blood solubility, potency, time required for induction and amount of gas required to saturate blood.

I am just a medical student and not a pro anesthesiologist or anything. If I have made a mistake or explained a concept wrong, lemme know!

Anteroinferior dislocation of shoulder injuries axillary nerve 

Cholestyramine

nakimedicalblog:

The most common side effect of cholestyramine is?

1) Flatulent diarrhoea
2) Constipation
3) Tooth decay
4) Itching
5) Black stools

It causes constipation. Also used in post-ileal resection Crohn’s patients.

The Crohn’s disease concept is so cool.

Cholestyramine is a bile acid sequestrant that binds bile salts, which are poorly absorbed in Crohn’s disease and might otherwise cause colonic irritations and diarrhea upon entering the colon.